Melatonin Plus: Melatonin – Wikipedia

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Melatonin Plus: Melatonin - Wikipedia

Melatonin – Wikipedia

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^ a b “Summary Safety Review – MELATONIN (N-acetyl-5-methoxytryptamine) – Review of the Melatonin in Children and Adolescents”. Government of Canada. Health Canada. 10 December 2015. Retrieved 9 January 2019.

^ a b “Melatonin- Oral”. Government of Canada. Health Canada. 28 August 2018. Retrieved 9 January 2019.

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^ Hardeland R (July 2005). “Antioxidative protection by melatonin: multiplicity of mechanisms from detoxification to radical avoidance”. Endocrine. 27 (2): 119–30. doi: 10.1385 / ENDO: 27: 2: 119. PMID 16217125.

^ Reiter RJ, Acuña-Castroviejo D, Tan DX, Burkhardt S (June 2001). “Free radical-mediated molecular damage. Mechanisms for the protective action of melatonin in the central nervous system.” Ann. N. Y. Acad. Sci. 939 (1): 200–15. Bibcode: 2001NYASA.939..200R. doi: 10.1111 / j.1749-6632.2001.tb03627.x. PMID 11462772.

^ Tan DX, Hardeland R, Manchester LC, Korkmaz A, Ma S, Rosales-Corral S, Reiter RJ (2012). “Functional roles of melatonin plants and plants and nutritional and agricultural science”. Journal of Experimental Botany. 63 (2): 577–97. doi: 10.1093 / jxb / err256. PMID 22016420.

^ “Australian Public Assessment Report for Melatonin” (PDF). www.tga.gov.au. The Australian Government Department of Health and Aging Therapeutic Goods Administration. January 2011. pp. 2, 4. Retrieved January 9, 2019. Monotherapy for patients at home.

^ McCarter SJ, et al. (March 2013). “Treatment Outcomes in REM Sleep Behavior Disorder”. Sleep Medicine (Review). 14 (3): 237–242. doi: 10.1016 / j.sleep.2012/09/09. PMC 3617579. PMID 23352028.

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^ McCleery J, Cohen DA, Sharpley AL (November 2016). “Pharmacotherapies for sleep disturbances in dementia”. The Cochrane Database of Systematic Reviews. 11: CD009178. doi: 10.1002 / 14651858.cd009178.pub3. PMID 27851868.

^ a b Herxheimer A, Petrie KJ (2002). “Melatonin for the Prevention and Treatment of Jet Lag”. Cochrane Database Syst Rev (2): CD001520. doi: 10.1002 / 14651858.CD001520. PMID 12076414.

^ a b Liira J, Verbeek JH, Costa G, Driscoll TR, Sallinen M, Isotalo LK, Ruotsalainen JH (August 2014). “Pharmacological interventions for sleepiness and sleep disturbances caused by shift work”. The Cochrane Database of Systematic Reviews. 8 (8): CD009776. doi: 10.1002 / 14651858.CD009776.pub2. PMID 25113164.

^ Buscemi N, Vandermeer B, Hooton N, Pandya R, Tjosvold L, Hartling L, Baker G, Klassen TP, Vohra S (2005). “The efficacy and safety of exogenous melatonin for primary sleep disorders. A meta-analysis”. J Gen Intern Med. 20 (12): 1151–58. doi: 10.1111 / j.1525-1497.2005.0243.x. PMC 1490287. PMID 16423108.

^ Buscemi N, Vandermeer B, Hooton N, Pandya R, Tjosvold L, Hartling L, Vohra S, Klassen TP, Baker G (February 2006). “Efficacy of exaturation for secondary sleep conditions and secondary restraints; BMJ. 332 (7538): 385–93. doi: 10.1136 / bmj.38731.532766.F6. PMC 1370968. PMID 16473858.

^ Lyseng-Williamson KA (2012). “Melatonin prolonged release in patients aged ≥55 years”. Drugs Aging. 29 (11): 911–23. doi: 10.1007 / s40266-012-0018-z. PMID 23044640.

^ Database of Reviews of Effects (DARE): Quality-assessed Reviews [Internet]. York (UK): Center for Reviews and Dissemination (UK); 1995. Optimal dosages for melatonin supplementation therapy in older adults: a systematic review of current literature. 2014

^ Brent Bauer, M.D. “Melatonin side effects: What are the risks?”. Mayo Clinic. Retrieved 17 August 2011.

^ Zhdanova IV, Wurtman RJ, Regan MM, Taylor JA, Shi JP, Leclair OU (October 2001). “Melatonin treatment for age-related insomnia”. J. Clin. Endocrinol. Metab. 86 (10): 4727-30. doi: 10.1210 / jc.86.10.4727. PMID 11600532.

^ Morera AL, Henry M, de la Varga M (2001). “Seguridad en el uso de la melatonina” [Safety in melatonin use]. Actas Esp Psiquiatr (in Spanish). 29 (5): 334–37. PMID 11602091.

^ Terry PD, Villinger F, Bubenik GA, Sitaraman SV (January 2009). “Melatonin and ulcerative colitis: evidence, biological mechanisms, and future research”. Inflamm. Bowel Dis. 15 (1): 134–40. doi: 10.1002 / ibd.20527. PMID 18626968.

^ Juszczak M, Michalska M (2006). “Wpływ melatoniny na syntezę i wydzielanie prolaktyny, hormonu luteinizującego (LH) i folikulotropowego (FSH)” [The effect of melatonin on prolactin, luteinizing hormone (LH), and follicle-stimulating hormone (FSH); Postepy Hig Med Dosw (Online) (in Polish). 60: 431–38. PMID 16921343.

^ Srinivasan V, Spence WD, Pandi-Perumal SR, Zakharia R, Bhatnagar KP, Brzezinski A (December 2009). “Melatonin and human reproduction: shedding light on the darkness hormone”. Gynecol. Endocrinol. 25 (12): 779–85. doi: 10.3109 / 09513590903159649. PMID 19905996.

^ Shane-McWhorter L. Melatonin. Merck Manual Professional Version. October 2018. Accessed 22 April 2019.

^ Emet, M; Ozcan, H; Ozel, L; Yayla, M; Halici Z; Hacimuftuoglu, A (June 2016). “A Review of Melatonin, Its Receptors and Drugs.” The Eurasian Journal of Medicine. 48 (2): 135–41. doi: 10.5152 / eurasianjmed.2015.0267. PMC 4970552. PMID 27551178.

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^ Ardura J, Gutierrez R, Andres J, Agapito T (2003). “Emergence and evolution of the circadian rhythm of melatonin in children”. Horm. Res. 59 (2): 66–72. doi: 10.1159 / 000068571. PMID 12589109.

^ Sack RL, Lewy AJ, Erb DL, Vollmer WM, Singer CM (1986). “Human melatonin production decreasing with age”. J. Pineal Res. 3 (4): 379–88. doi: 10.1111 / j.1600-079X.1986.tb00760.x. PMID 3783419.

^ Hagenauer MH, Perryman JI, Lee TM, Carskadon MA (June 2009). “Adolescent changes in the homeostatic and circadian regulation of sleep”. Developmental Neuroscience. 31 (4): 276–84. doi: 10.1159 / 000216538. PMC 2820578. PMID 19546564.

^ Tan DX, Chen LD, Poeggeler B, L Manchester C, Reiter RJ. Melatonin: a potent, endogenous hydroxyl radical scavenger. Endocr. J. 1993, 1, 57–60 https://docs.google.com/viewer?a=vpid=sitessrcid=ZGVmYXVsdGRvbWFpbnxkdW54aWFudGFufGd4OjVkMjA5NGZkMzFmYjRkOTU

^ Poeggeler B, Saarela S, Reiter RJ, Tan DX, Chen LD, Manchester LC, Barlow-Walden LR (November 1994). “Melatonin – a highly potent endogenous radical in vitro”. Ann. N. Y. Acad. Sci. 738 (1): 419–20. Bibcode: 1994NYASA.738..419P. doi: 10.1111 / j.1749-6632.1994.tb21831.x. PMID 7832450.

^ a b Arnao MB, Hernández-Ruiz J (May 2006). “The physiological function of melatonin in plants”. Plant Signal Behav. 1 (3): 89–95. doi: 10.4161 / psb.1.3.2640. PMC 2635004. PMID 19521488.

^ Pieri C, Marra M, Moroni F, Recchioni R, Marcheselli F (1994). “Melatonin: a peroxyl radical scavenger more effective than vitamin E”. Life Sci. 55 (15): PL271–76. doi: 10.1016 / 0024-3205 (94) 00666-0. PMID 7934611.

^ a b c Sharafati-Chaleshtori R, Shirzad H, Rafieian-Kopaei M, Soltani A (2017). “Melatonin and human mitochondrial diseases”. Journal of Research in Medical Sciences. 22: 2. doi: 10.4103 / 1735-1995.199092. PMC 5361446. PMID 28400824.

^ a b c d e f g h Jockers R, Delagrange P, Dubocovich ML, Markus RP, Renault N, Tosini G, Cecon E, Zlotos DP (September 2016). “Update on melatonin receptors: IUPHAR Review 20”. British Journal of Pharmacology. 173 (18): 2702–25. doi: 10.1111 / bph.13536. PMC 4995287. PMID 27314810. It makes it possible to increase the amount of reactive species, such as the vitamin C and vitamin E, under in vitro or in vivo conditions (Gitto et al., 2001; Sharma and Haldar, 2006; Ortiz et al., 2013).

^ a b c Reiter RJ, Rosales-Corral S, Tan DX, Jou MJ, Galano A, Xu B (November 2017). “Melatonin as a mitochondria-targeted antioxidant: one of evolution’s best ideas.” Cellular and Molecular Life Sciences. 74 (21): 3863–3881. doi: 10.1007 / s00018-017-2609-7. PMID 28864909. Melatonin is specifically targeted to the mitochondria where it seems to be the apox antioxidant …

^ a b c Reiter RJ, Mayo JC, Tan DX, Sainz RM, Alatorre-Jimenez M, Qin L (October 2016). “Melatonin as an antioxidant: under promises but over delivers”. Journal of Pineal Research. 61 (3): 253–278. doi: 10.1111 / jpi.12360. PMID 27500468. Mitochondria-targeted antioxidant.

^ a b c Manchester LC, Coto-Montes A, Boga JA, Andersen LP, Zhou Z, Galano A, Vriend J, Tan DX, Reiter RJ (November 2015). “Melatonin: an oxygen molecule that makes oxygen metabolically tolerable.” Journal of Pineal Research. 59 (4): 403–19. doi: 10.1111 / jpi.12267. PMID 26272235. It’s now known as the pineal gland [53] have been investigated [48, 55, 56]. It is not relegated solely to the pineal gland [57–59], it is not relegated.

^ a b c d e f Mayo JC, Sainz RM, González-Menéndez P, Hevia D, Cernuda-Cernuda R (November 2017). “Melatonin transport into mitochondria”. Cellular and Molecular Life Sciences. 74 (21): 3927–3940. doi: 10.1007 / s00018-017-2616-8. PMID 28828619.

^ Carrillo-Vico A, Guerrero JM, Lardone PJ, Reiter RJ (July 2005). “A review of the multiple actions of the melatonin on the immune system.” Endocrine. 27 (2): 189–200. doi: 10.1385 / ENDO: 27: 2: 189. PMID 16217132.

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Melatonin Plus: Melatonin - Wikipedia

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^ a b c Tan DX, Hardeland R, Manchester LC, Korkmaz A, Ma S, Rosales-Corral S, Reiter RJ (January 2012). “Functional roles of melatonin plants and plants and nutritional and agricultural science”. Journal of Experimental Botany. 63 (2): 577–97. doi: 10.1093 / jxb / err256. PMID 22016420.

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